Researchers have found that short-chain fatty acids have protective effects in early-onset type 1 diabetes
November 09, 2018 Source: Ministry of Science and Technology
Window._bd_share_config={ "common":{ "bdSnsKey":{ },"bdText":"","bdMini":"2","bdMiniList":false,"bdPic":"","bdStyle":" 0","bdSize":"16"},"share":{ }};with(document)0[(getElementsByTagName('head')[0]||body).appendChild(createElement('script')) .src='http://bdimg.share.baidu.com/static/api/js/share.js?v=89860593.js?cdnversion='+~(-new Date()/36e5)];On October 24th, researchers at the Broad Institute of MIT and Harvard in the United States published a book entitled "The human gut microbiome in early-onset type 1 diabetes from the TEDDY study" in the journal Nature. In the Journal of Juvenile Diabetes Environmental Determinants (TEDDY), the gut microbiota was analyzed in patients with early-onset type 1 diabetes.
Type 1 diabetes (T1D) is an organ-specific autoimmune disease characterized by targeted destruction of islet β-cells. The pathogenesis involves the interaction of genetic and environmental factors, viral infection and comparison. A wide range of intestinal diseases have been identified as potential causes or pathogenic factors of T1D. The researchers analyzed 10,913 metagenomic samples from 783 children (mainly white, non-Hispanic) stool samples and found that the microbes in the control group contained more genes related to biosynthesis and fermentation of short-chain fatty acids. . Combining the available evidence from human cohort studies and T1D mouse model studies, this study demonstrates that short-chain fatty acids have protective effects in early-onset T1D. The analysis of the intestinal genome in the TEDDY study laid the foundation for the study of intestinal microbiota and islet autoimmunity, T1D and other early childhood diseases in children's growth and development.
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